By Sonja M. Lillrank
Examines the indications, reasons, and results of Alzheimer's sickness and different dementias and demonstrates how numerous remedies have an effect on the mind to assist deal with those disorders...Title: .Alzheimer's affliction And different Dementias..Author: .Lillrank, sonja M., M.d./ Collins, Christine Collins, Ph.d. (EDT)/ Levitt, Pat (FRW)..Publisher: .Facts on File..Publication Date: .2007/01/01..Number of Pages: .118..Binding sort: .LIBRARY..Library of Congress: .2006010414
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Additional info for Alzheimer's Disease and Other Dementias
These cognitive problems reflect damage to the cortex of the brain, which processes information from other parts of the brain as well as from various parts of the body and peripheral nervous system. Finally, the patient’s problems must interfere with social and occupational functioning to qualify as dementia. MEMORY Memory problems are often the first sign of dementia. It can be difficult to tell the difference between normal age-related memory problems and those caused by dementia. Examples of normal forgetfulness would be forgetting what you were supposed to buy at the store or having problems remembering phone numbers.
Other blood tests may be indicated based on the patient’s family and medical history. Often a brain scan like computed tomography (CT) or magnetic resonance imaging (MRI) is performed to rule out strokes, tumors, or normal pressure hydrocephalus (NPH). In NPH, increased pressure in the brain and enlargement of the fluid-filled cavities in the brain cause specific dementia-like symptoms. Sometimes a brain scan will verify the NPH dementia diagnosis, especially if changes like cortical atrophy, or withering of brain cells on the cortex (the surface of the brain), indicates that the brain has shrunk.
These plaques are most prominent in the brain regions that are important for memory, thinking, and decision-making. These regions include the hippocampus and temporal and parietal regions of the cerebral cortex. It is not clear what the normal function of APP is, but it appears that it may be important in helping neurons to grow and survive. It is also not clear whether the beta-amyloid plaques themselves cause Alzheimer’s disease or whether they are just a byproduct of the illness. If the beta-amyloid plaques indeed are the cause of Alzheimer’s disease, then it might be possible to treat patients by stimulating the immune system to destroy the beta-amyloids, or by inhibiting the secretase enzymes that cut the APP into beta-amyloid fragments.
Alzheimer's Disease and Other Dementias by Sonja M. Lillrank